Adjusting for Publication Bias Reveals Evidence Against Social Comparison As a Behaviour Change Technique Across the Behavioural Sciences
Frantisek Bartos, Zuzana Irsova, Tomas Havranek, Eric-Jan Wagenmakers (2025), "Adjusting for Publication Bias Reveals Evidence Against Social Comparison As a Behaviour Change Technique Across the Behavioural Sciences," available at osf.io/preprints/psyarxiv/jtv5d_v1. https://doi.org/10.31234/osf.io/jtv5d_v1.
František Bartoš1,2*, Zuzana Iršová2, Tomáš Havránek,2,3, Eric-Jan Wagenmakers1
1 Department of Psychological Methods, University of Amsterdam
2 Institute of Economic Studies, Faculty of Social Sciences, Charles University
3 Meta-Research Innovation Center at Stanford
Correspondence concerning this article should be addressed to: František Bartoš, University of Amsterdam, Department of Psychological Methods, Nieuwe Achtergracht 129-B, 1018 VZ, Amsterdam, The Netherlands
f.bartos96@gmail.com
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Adjusting for Publication Bias Reveals Evidence Against Social Comparison As a Behaviour Change Technique Across the Behavioural Sciences
Social comparison is increasingly used as a behaviour change technique (SC-BCT) yet its true efficacy remains unclear. To address this concern, Hoppen and colleagues1 performed a comprehensive meta-analysis of 79 randomized controlled trials involving over 1.3 million participants. Hoppen and colleagues1 “found evidence supporting the efficacy of SC-BCTs in shaping behaviour in the desired direction”. However, their own risk of bias assessment revealed that “most trials (k = 66; 84%) had some concern of bias regarding selection of the reported result(s) given that preregistrations and prespecified analysis protocols were rare” and they noted that “it remains unknown to what extent the present results are affected by publication bias”. In our re-analysis, we demonstrate that publication bias exaggerated the evidence in favor of SC-BCT to such a degree that once this bias is properly adjusted for, the effect disappears entirely. In fact, the data show moderate evidence against the presence of an effect.
For any meta-analysis it is crucial to report estimates that have been properly adjusted for publication bias–the suppression of non-significant and non-conforming results–, since this bias can inflate effect sizes and produce spurious evidence in favor of an effect2,3. Hoppen and colleagues1 did test for publication bias using Egger’s regression, and they applied trim and fill publication bias adjustments in cases where Egger’s test was statistically significant. For comparisons with passive control conditions, Egger’s test failed to reject the null hypothesis of no publication bias; for comparisons with active controls, Egger’s test did indicate publication bias, but the trim and fill adjustment had little impact on the estimated effect size. Unfortunately, Egger’s test and trim and fill are known to perform poorly in detecting4,5 and adjusting2,6,7 for publication bias in the presence of heterogeneity, a scenario that applies to the meta-analysis by Hoppen and colleagues1.
We re-examine the Hoppen and colleagues1 data using robust Bayesian meta-analysis8. RoBMA accounts for publication bias by combining a set of selection models9 and PET-PEESE10, a meta-regression correction technique based on the funnel plot, via Bayesian model-averaging11. These methods have been shown to greatly outperform publication-bias unadjusted meta-analyses in both simulation studies4,5,12 and real data scenarios2,12. Furthermore, Bayesian model averaging allows RoBMA to place more weight on models that predict the data well, making it more robust to model misspecification and directly evaluate the evidence for the presence vs. absence of the effect, heterogeneity, and publication bias via Bayes factors13 (rather than absence of evidence with non-significant p-values).
Figure 1 shows the RoBMA meta-analytic publication bias-adjusted estimates for the efficacy of SC-BCT in comparison to passive and active control conditions. After adjusting for publication bias, the efficacy of SC-BCT in comparison to passive control condition decreases from g = 0.17, 95% CI [0.11, 0.23] to g = 0.00, 95% CI [-0.11, 0.04] and the efficacy of SC-BCT in comparison to active control conditions decreases from g = 0.23, 95% CI [0.15, 0.31] to g = 0.01, 95% CI [0.00, 0.14]. In essence, the efficacy of SC-BCT completely disappears once publication bias adjustment is performed. Moreover, the evidence for the presence of the effects turns into evidence against the effects, with the data presenting moderate evidence against the effect in comparisons of SC-BCT with passive conditions, BF10 = 0.108 (i.e., BF01 = 9.3), and moderate evidence against the effect in comparisons of SC-BCT with active conditions, BF10 = 0.165 (i.e., BF01 = 6.1). This means that the observed data are about 9.3 and 6.1 times as likely to occur under the models that assume the absence of the effects than under the models that assume their presence. This reversal in conclusions results from the publication bias detected by and adjusted for by RoBMA; RoBMA shows extreme evidence for the presence of publication bias in both the comparison of SC-BCT with passive control conditions, BF10 = 450, and active control conditions, BF10 = 3,999.
Table 1 summarizes the results of a sensitivity analysis with a set of selection models and PET-PEESE individually for all main results reported by Hoppen and colleagues1. This sensitivity analysis of the short-term efficacy demonstrates that while some of the results differ based on the publication bias adjustment selected, the conclusions are leaning towards the absence of SC-BCT efficacy. The sensitivity analysis of the follow-up effects is much more complicated since the low number of estimates limits the feasibility of publication bias adjustment. Importantly, the individual models included in the sensitivity analysis should not be overinterpreted in isolation, as some might be poorly suited for data with substantial heterogeneity and would receive little weight in the Bayesian model-averaging approach.
Our reanalysis suggests that the apparent efficacy of SC-BCT in shaping behaviour in the desired direction reported by Hoppen and colleagues1 is largely a byproduct of publication bias. Once publication bias is properly adjusted for, the efficacy of SC-BCT disappears and the data show moderate evidence against the presence of the effect. These findings align with previous publication bias corrected estimates of short term behavioural interventions14,15, which raises the question about the feasibility and effectiveness of simple behavioural change techniques.
Note. RoBMA model-averaged posterior mean effect size estimates with 95% credible intervals and Bayes factors for the presence of the effect for each outcome. BF10 quantifies evidence for the alternative hypothesis. BF10 larger than 1 corresponds to evidence in favour of the alternative hypothesis, and BF10 lower than 1 corresponds to evidence in favour of the null hypothesis (evidence for the alternative hypothesis can be obtained by inverting the Bayes factor; BF10 = 1/BF01). As a rule of thumb, Bayes factors between 3 and 10 indicate moderate evidence, and Bayes factors larger than 10 indicate strong evidence. Figure from JASP.
| RoBMA | Selection Model (step at p = 0.025) | Selection Model (step at p = 0.025 and 0.50) | PET-PEESE | |
|---|---|---|---|---|
| Post-intervention results: short-term efficacy | ||||
| SC-BCTs versus passive control conditions | 0.00 [-0.11, 0.04] | 0.20 [0.10, 0.29] | -0.05 [-0.37, 0.28] | 0.21 [0.14, 0.28] |
| BF10 = 0.108 | p < 0.001 | p = 0.778 | p < 0.001 | |
| SC-BCTs versus passive control conditions (outlier-adjusted) | -0.01 [-0.17, 0.02 ] | 0.20 [0.11, 0.29] | -0.06 [-0.40, 0.28] | 0.21 [0.14, 0.29] |
| BF10 = 0.122 | p < 0.001 | p = 0.734 | p < 0.001 | |
| SC-BCTs versus active control conditions | 0.01 [0.00, 0.14] | 0.27 [0.13, 0.41] | 0.15 [-0.07, 0.36] | 0.03 [-0.00, 0.06] |
| BF10 = 0.165 | p < 0.001 | p = 0.175 | p = 0.058 | |
| SC-BCTs versus active control conditions (outlier-adjusted) | 0.00 [0.00, 0.15] | 0.25 [0.13, 0.38] | 0.14 [-0.05, 0.33] | 0.03 [0.00, 0.06] |
| BF10 = 0.199 | p < 0.001 | p = 0.150 | p = 0.051 | |
| Follow-up results: long-term efficacy | ||||
| SC-BCTs versus passive control conditions | 0.03 [0.00, 0.12] | 0.10 [0.06, 0.13] | 0.10 [0.07, 0.14] | 0.09 [0.03, 0.15 |
| BF10 = 0.638 | p < 0.001 | p < 0.001 | p = 0.014 | |
| SC-BCTs versus passive control conditions (outlier-adjusted) | 0.06 [0.00, 0.12] | 0.10 [0.07, 0.14] | 0.11 [0.07, 0.14] | 0.11 [0.07, 0.14] |
| BF10 = 1.928 | p < 0.001 | p < 0.001 | p < 0.001 | |
| SC-BCTs versus active control conditions | 0.00 [0.00, 0.00] | 0.71 [-0.02, 1.43] | Non-estimable | 0.01 [-0.00, 0.03] |
| BF10 = 0.030 | p = 0.056 | p = 0.155 | ||
Note. RoBMA model-averaged posterior mean effect size estimates with 95% credible intervals and Bayes factors for the presence of the effect for each outcome. Selection models (either one or two-step selection on one-sided p-values) and PET-PEESE with effect size estimate with 95% confidence interval and p-value against the null hypothesis of no effect.
Data & Materials
The data and analysis script are available at https://osf.io/rj2g5/.
Acknowledgements
František Bartoš and Zuzana Iršová acknowledge support from the Czech Science Foundation (grant 23-05227M). Tomáš Havránek acknowledges support from the Czech Science Foundation (grant 24-11583S) and from the Institute for Research on the Socioeconomic Impact of Diseases and Systemic Risks (grant LX22NPO5101), funded by the European Union–Next Generation EU.
Competing interests
Authors declare no competing interests.
Author Contribution
FB analysed the data and wrote the first draft of the manuscript, all authors edited and approved the final version of the manuscript.
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